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Accelerated Atherosclerosis in Apoe−/− Mice Heterozygous for the Insulin Receptor and the Insulin Receptor Substrate-1 | Arteriosclerosis, Thrombosis, and Vascular Biology
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Cells | Free Full-Text | Piezo1 Channel Activation Reverses Pulmonary Artery Vasoconstriction in an Early Rat Model of Pulmonary Hypertension: The Role of Ca2+ Influx and Akt-eNOS Pathway | HTML
PDF) Multiple Inducers of endothelial NOS (eNOS) Dysfunction in Sickle Cell Disease
Cells | Free Full-Text | Piezo1 Channel Activation Reverses Pulmonary Artery Vasoconstriction in an Early Rat Model of Pulmonary Hypertension: The Role of Ca2+ Influx and Akt-eNOS Pathway | HTML
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The role of androgen receptors in atherosclerosis - ScienceDirect
Assessing the functional properties of high-density lipoproteins: an emerging concept in cardiovascular research | Biomarkers in Medicine
Cells | Free Full-Text | Endothelial Dysfunction Following Enhanced TMEM16A Activity in Human Pulmonary Arteries | HTML
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Contraction by Ca2+ Influx via the L-Type Ca2+ Channel Voltage Window in Mouse Aortic Segments is Modulated by Nitric Oxide | IntechOpen
Unexpected Effect of Proton Pump Inhibitors | Circulation
Unexpected Effect of Proton Pump Inhibitors | Circulation
Cells | Free Full-Text | Piezo1 Channel Activation Reverses Pulmonary Artery Vasoconstriction in an Early Rat Model of Pulmonary Hypertension: The Role of Ca2+ Influx and Akt-eNOS Pathway | HTML
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Cells | Free Full-Text | Endothelial Dysfunction Following Enhanced TMEM16A Activity in Human Pulmonary Arteries | HTML
Multiple inducers of endothelial NOS (eNOS) dysfunction in sickle cell disease - Hebbel - 2021 - American Journal of Hematology - Wiley Online Library
Contraction by Ca2+ Influx via the L-Type Ca2+ Channel Voltage Window in Mouse Aortic Segments is Modulated by Nitric Oxide | IntechOpen